acknowledges the role of biological factors - abnormal brain activity producing visual and auditory hallucinations
Further features emerge as people try to make sense of the hallucinations
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AO1 2 - Cognitive explanation
First experience worrying sensory experiences - turn to others to confirm the validity
other people fail to confim - believe others are hiding the truth
Reject feedback from others and develop delusional beliefs that they are being manipulated and persecuted by others.
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AO1 3 Life events
Stressful life events cause the onset of SZ e.g. death of a close relative act as a trigger
Individual may have a biological predispostition for SZ but only some people with such will develop the disorder - those who experience stressors
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AO1 Brown and Birley
Prior to a SZ episode, patients reported twice as many stressful life events compared to a healthy control group - low and unchanging level of stressful life events
Illustrates the link
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AO2 1 Biological evidence
Meyer Lindberg - link between excess levels of dopamine in the prefrontal cortex and dysfunctions in working memory
Working memory dysfunction is associated with the cognitive disorganisation typically found in SZ.
biological factors underlie some of the early symptoms of SZ
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IDA Therapy
Yellowlees - trialled a machine that can deliever 'virtual auditory and virtual hallucinations.'
Intention to show SZ that their hallucinations are not real.
However, no evidence that this will provide a successful treatment
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AO2 2 Retrospective
Brown and Birley's research is retrospective - data collected after events have occured
Once a person has developed SZ asked questions about events leading upto the onset.
Recall would be negatively affected by events surrounding the onset - unreliable
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AO2 3 Hirsch et al
Prospective studies are preferable - studied after the onset of the disorder
Hirsch - followed 71 SZ patients over a 48 week period.
Clear that life events made a cumulative contribution in the 12 months preceeding relapse rather than having a more concentrated effect in the period just prior to the episode.
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AO2 4 Van Os et al
Not all research supports life events
Van Os et al - no link between life events and the onset of SZ.
Prospective part - patients who had experienced a major life event went on to have a lower incidence of relapse.
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AO2 5 Correlational
research is correlational
cannot infer a causal relationship between stressful life events and SZ
early symptoms were the cause of life events e.g. divorce
may be a consequence rather than a cause
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IDA Approaches
Biological explanations have better research support
large body of evidence - role of genetic factors
Gottesman - greater degree of relatedness the greater risk of SZ
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