1. Activation of the nicotinic type II receptors causes what?
- Opening of voltage-gated sodium channels
- Activation of G-protein coupled calcium-dependent receptor
- Opening of voltage-gated calcium channels
- Activation of G-protein coupled sodium-dependent receptor
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2. Through what mechanism is ACh released from the presynaptic membrane?
- Sodium-dependent endocytosis
- Calcium-dependent endocytosis
- Calcium-dependent exocytosis
- Sodium-dependent exocytosis
3. Flacced paralysis is an eefect of denervation. Which of the following best describes flaccid paralysis?
- Redistribution of ACh receptors away from motor end plates causing reactivity to ACh along the length of the muscle fibre
- No voluntary or involuntary muscle contraction, no resting muscle tone and no active resistance to stretch
- Muscle wasting, loss of contractile protein
- Spontaneous twitching of motor units due to denervation hypersensitivity
4. How many sub-units make up the nicotinic type II receptors?
5. What is an end-plate potential?
- Depolarization of the post-synaptic terminal
- Depolarization of the motor end plate
- Repolarization of the motor end plate
- Repolarization of the post-synaptic terminal
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