TB6 B&B Lecture 1 MCQ; Object recognition

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  • Created by: mint75
  • Created on: 26-11-15 14:34
What did Newcombe's (1987) double dissassociation finding show?
Pps with right parietal lobe lesions performed poorly on spatial tasks (maze) and well on face/object recognition, whereas pps with right temporal lobe lesions performed opposite
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From V1, where does the dorsal stream broadly terminate?
Posterior parietal cortex
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What happens to the size of the receptive fields of ventral stream neurons across the stream?
The receptive field size increases and responses shift towards more complex features
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What is the general assumption behind fMRI adaptation methods?
Due to neural responses to constant stimulus presentation decreasing over time, it can be ascertained what makes a stimulus 'different'/same for a population of neurons
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What did Kourtzi & Kanwishers fMR adaptation study find specifically for the (bi)lateral occipital cortex?
Responses were contrasted to scrambled v.s intact object images. The LOC showed to adapt to the same shape with different occluding contours
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What is visual agnosia?
A deficit in visual recognition in the absence of deficits in sensory functions (e.g vision), can be modality-specific
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What is apperceptive agnosia?
Deficits at a perceptual level. Patients often are better at drawing from memory rather than copying an object's form directly and have preserved colour/brightness perception
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What is associative/integrative agnosia?
Deficits at the attribution level, where the percept is given meaning by being linked to previous experience. There is accurate copying from vision, but poor drawing from memory
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From Milner & Goodale (1965), what were patients DF's deficits?
Damaged ventral stream. (Apperceptive agnosia) Could not say whether two shapes were the same or not. Could adjust aperture of fingers correctly when reaching for objects.
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From Humphrey & Riddoch (1987), what were patient HJAs neural deficits?
Bilateral damage to ventral visual stream. Achromatopsia. Could not recognise faces. Very poor OR, but could be achieved with feature-by-feature analysis. Could not segment/group features
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From Humphrey & Riddoch (1987), what were patient HJAs behavioural abilities?
Can copy drawings of objects (feature by feature), can draw from memory + can recognise from other modalities. Cannot recognise pictures even though can describe features, cannot use gestalt grouping principles, cant decide if novel objects are real
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What was the double dissassociation finding from Goodale et al (1994) with patient RV and case DF?
Case RV (damage to dorsal) could tell the shape of objects apart but could not correctly grasp them. DF (ventral damage) performed at chance at form discrim but could successfully grasp
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What was the key point made by Goodale and Humphrey (1998) re; organisational principles?
The underlying and essential processes behind vision for perception and vision for action are highlighted by their placement in the brain
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From V1, where does the dorsal stream broadly terminate?

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Posterior parietal cortex

Card 3

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What happens to the size of the receptive fields of ventral stream neurons across the stream?

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Card 4

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What is the general assumption behind fMRI adaptation methods?

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Card 5

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What did Kourtzi & Kanwishers fMR adaptation study find specifically for the (bi)lateral occipital cortex?

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