MEMORY PSYCHOLOGY

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EXPLICIT
SCHACTER forgetting in amnesiacs down to imapared explicit memory.amnesiacs better in implicit than explicit tasks. eg HM in gollin test, improved through repetition and remembered part of images (never remembered doing the test). draw using a mirror
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DIFFERENT LTMs
mirror drawing indicates different LTMs. Procedural memory= phsyical skill, Declarative memory= fact related.amnesiacs bad explicit memory and good implicit. perform good procedural, bad declarative. HM drawing shows difference
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STRENGTHS AND LIMITATIONS
STICKGOLD tetristo test memory. normal people improve at it and able to describe, amnesiacs improve slowly but no recall or explain. shows implicit development. SCHACTER presenting learned words-pairs same voice helped 'normals', didnt help amnesiacs
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RATIONAL MEMORY BINDING
implicit/explicit memory show differences in mem function of amnesiacs but doesnt explain why(descriptive not explanatory).explained by RMB (amnesiacs lack function that links pieces of info.(cannot see relationship between exp/imp aspects of events)
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ANTEROGRADE
Anterograde amnesia (type 1) problems with creating new LTMs. ISAAC AND MAYES say problem with consolidation or retrieval,tested amnesiacs and control, amnesiacs performed good cue recall and recognition. shows problem with consoldiation
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RETROGRADE
retrograde (type 2) loss of past event memory. retrograde amnesia on onset of disease. Memory older than onset imporves over time.Temporal gradiant (chnage over time) shows LTMs need to be consolidated or 'lost'.symptom caused by disruption process
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BRAINSCAN
some have retro and antero (both caused by problems in same area).GABRIELI damage to part of hippo cause antero alone.REED AND SQUIRE mri 4 p's all had hippo damage, worst had temperal lobe damage.REMONDES & SCHMAN rats damage hippo leanred maze but
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B-AMYLOIDS
alzheimers memory loss- b-amyloids/ plaques. amylod precursor protein in brain break down to produce b-amyloid protein 40.(in AD sufferes it prodecuses B-amyloid protie 42) builds up between neuron plaques=communication problems.
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PLAQUES
plaque buildup = CerCort to shrink,causingfolds on its surface enlarge.hippo also affected.change affect memory =basal forebrain (arousal in CerCort) is damaged=less alert,hard to attend stimuli. SNYDER ET AL B-amy affects NMDA (neuroT of learning)
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TANGLES
AD change in brain=development of tangles.= build up of tall protein (normally provides support for cell structure within neuron). tangles hppen when structure of neuron's cell body degenerates
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Card 2

Front

mirror drawing indicates different LTMs. Procedural memory= phsyical skill, Declarative memory= fact related.amnesiacs bad explicit memory and good implicit. perform good procedural, bad declarative. HM drawing shows difference

Back

DIFFERENT LTMs

Card 3

Front

STICKGOLD tetristo test memory. normal people improve at it and able to describe, amnesiacs improve slowly but no recall or explain. shows implicit development. SCHACTER presenting learned words-pairs same voice helped 'normals', didnt help amnesiacs

Back

Preview of the back of card 3

Card 4

Front

implicit/explicit memory show differences in mem function of amnesiacs but doesnt explain why(descriptive not explanatory).explained by RMB (amnesiacs lack function that links pieces of info.(cannot see relationship between exp/imp aspects of events)

Back

Preview of the back of card 4

Card 5

Front

Anterograde amnesia (type 1) problems with creating new LTMs. ISAAC AND MAYES say problem with consolidation or retrieval,tested amnesiacs and control, amnesiacs performed good cue recall and recognition. shows problem with consoldiation

Back

Preview of the back of card 5
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