Psychopathology Revision Cards
- Created by: millywhitehouse
- Created on: 28-12-20 11:44
AO1 Definitions of Abnormality: Statistical Infreq
Statistics describe typical values
A frequency graph of behaviours tends to show a normal distribution
The extreme ends define what is not the norm i.e abnormal
AO3 Definitions of Abnormality: Statistical Infreq
Some behaviour is desirable - can't distinguish from desirable and undesirable abnormal behaviour
Cut off point is subjective - important for deciding who gets treatment
Sometimes appropriate - e.g for intellectual disability define as less then two standards deviations below mean IQ
Cultural relativism - statistical infrequency is relative to the reference population
AO1 Definitions of Abnormality: Deviation from So
Norms defined by a group of people
Standards of what is acceptable
May be implicit or defined by law
AO3 Definitions of Abnormality: Deviation from Soc
Susceptible to abuse - varies with changing attitude/morals, can be used to incarcerate those who are nonconformists
Related to context and degree - e.g shouting is normal in some places and in moderation
Strengths - distinguishes desirable from nondesirable behaviour, and considers the effects on others
Cultural relativism - social norms of dominant culture used as basis for DSM, imposed on other cultural groups
AO1 Definitions of Abnormality: Failure to Functio
Being unable to manage everyday life e.g eating regularly
Lack of functioning is abnormal if it causes distress to self and/or others
WHODAS used to provide quantitative measure of functioning
AO3 Definitions of Abnormality: Failure to Functio
Distress may be judged subjectively
Behaviour may be functional e.g depression may be rewarding for the individual
Strengths - recognised subjective experience for the individual can be measure objectively
Cultural relativism - standards of everyday life vary between cultures, non-traditional lifestyles may be judged as inadequate
AO1 Definitions of Abnormality: Deviation from Ide
Jahoda identified characteristics commonly used when describing competent people
For example, high self esteem, self actualisation, autonomy, accurate perception of reality, mastery of the environment
AO3 Definitions of Abnormality: Deviation from Ide
Unrealistic criteria - may not be useably because too ideal
Equates mental and physical health - whereas mental disorders tend not to have physical causes
Positive approach - a general part of the humanistic approach
Culture bound criteria e.g self-actualisation not relevant to collectivist cultures
Mental Disorders: Phobias
Emotional: excessive fear, anxiety and/or panic cued by a specific object or situation
Behavioural: avoidance, faint or freeze, interferes with everyday life
Cognitive: not helped by rational argument, unreasonableness of the behaviour recognised
Mental Disorders: Depression
Emotional: negative emotions - sadness, loss of interest and sometimes anger
Behavioural: reduced or increased by activity related to energy levels, sleep and/or eating
Cognitive: irrational, negative thoughts and self-beliefs that are self fulfilling
Mental Disorders: OCD
Emotional: anxiety and distress, and awareness that this is excessive, leading to shame
Cognitive: recurrent, intrusive, uncontrollable thoughts (obsessions), more than everyday worries
Behavioural: compulsive behaviours to reduce obsessive thoughts, not connected in a realistic way
AO1 BeA: Explaining Phobias
(The Two Process Model)
Classical conditioning - phobia acquired through association between NS and UCR, NS becomes CS, producing fear
Little Albert (Watson and Rayner) - developed fear of white rat which generalised to other white furry objects
Operant conditioning - phobia maintained through negative reinforcement (avoidance of fear)
Social learning - phobic behaviours of others modelled
AO3 BeA: Explaining Phobias
Classical conditioning - people often report a specific incident but not always, may only apply to some types of phobias (Sue et al)
Diathesis-stress model - not everyone bitten by a dog develops a phobia (di Nardo et al), may depend on genetic vulnerability for phobias
Social learning - fear response acquired through observing reaction to a Buzzer (Bandura and Rosenthal)
Biological preparedness - phobias more likely with ancient fears, conditioning alone can't explain all phobias
Two process model ignores cognitive factors - irrational thinking may explain social phobias
AO1 BeA: Treating Phobias
(Systematic De-sensitisation)
Counterconditioning - phobic stimulus associated with new response of relaxation
Reciprocal inhibition - the relaxation inhibits anxiety (Wolpe)
Relaxation - deep breathing, focus on peaceful scene, progressive muscle relaxation
Desenitisation hierarchy - from least to most fearful, relaxation practised at every step
AO3 BeA: Treating Phobias
Effectiveness - 75% success (McGrath et al) , in vivo techniques may work better or a combination (Comer)
Not for all phobias - works less well for ancient fears (Ohman et al)
Strengths - behavioural therapies are fast and require less effort than CBT, can be self administered
AO1 CA: Explaining Depression
(Ellis' ABC Model 1962)
Activating event leads to rational or irrational belief, which then leads to consequences
Mustabatory thinking causes disappointment and depression
(Beck's negative triad)
Negative schema - develops in childhood, lead to cognitive biases
Negative triad - irrational and negative view of self, the world and the future
AO3 CA: Explaining Depression
Support for role of irrational thinking - depressed people make more errors in logic (Hammen and Krantz) however, irrational thinking may not cause depression
Blames the client and ignores situational factors - recovery may depend on recognising environmental factors
Practical applications to CBT - support the role of irrational thoughts in depression
Irrational beliefs may be realistic - depressed people may be realists, 'sadder but wiser' (Alloy and Abrahamson)
Alternative explanation - genes may cause low levels of serotonin, predisposing people to develop depression
AO1 CA: Treating Depression
Ellis' ABCDEF model
D for disrupting irrational beliefs e.g logical, empirical, pragmatic
E and F for Eddecrs of disrupting and Feelings that are produced
Homework - trying out new behaviours to test irrational beliefs
Behavioural activation - encouraging re-engagement with pleasurable activities
Unconditional positive regard - reduces sense of worthlessness
AO3 CA: Treating Depression
Research support - generally successful, Ellis estimated 90% success over 27 sessions. May depend on therapist competence (Kuyken and Tsivirkos)
Indiviudal differences - CBT not suitable for those with rigid irrational beliefs, those whose stressors cannot be changed and those who don't want direct advice.
Behavioural activation - depressed clients in an exercise group has lower relapse after 6 months (Babyak et al)
Alternative treatments - drug therapy is much easier in time and effort, can be used with CBT
Dodo bird effect - all treatments equally effective because they share feature e.g talking to a sympathetic person (Rosenzweig)
AO1 BiA: Treating Phobias
(Flooding)
One long session with the most fearful stimulus
Continues until anxiety subsides and relaxation is complete
Can be in vivo or virtual reality
AO3 BiA: Treating Phobias
Individual differences - traumatic and, if the patients quit, then has failed as a treatment
Effectiveness - research suggests it may be more effective than SD and quicker (Choy et al)
Relaxation may not be necessary - creating a new expectation of coping may matter more (Klein et al)
Symptom substitution - a phobia may be a symptom of an underlying problem (e.g Little Hans)
AO1 BiA: Explaining OCD - Genetic Explanations
COMPT gene - one allele more common in OCD creates high levels of dopamine (Tukel et al)
SERT gene- one allele more common in a family with OCD, creates low levels of serotonin
Diathesis-stress - same genes linked to other disorders or no disorder at all, therefore, genes create a vulnerability
AO1 BiA: Explaining OCD - Neural Explanations
Dopamine levels high in OCD - linked to compulsive behaviours in animal studies
Serotonin levels low in OCD - antidepressants that increase serotonin are effective
Worry circuit - damaged caudate nucleus doesn't suppress worry signals from OFC to thalamus
Serotonin and dopamine linked to activity in these parts of the frontal lobe
AO3 BiA: Explaining OCD
Studies of first-degree relatives - 5 times greater risk of OCD if relative has OCD (Nestadt et al)
Twin studies - twice as likely to have OCD if MZ twins (Billett et al)
Environmental component - concordance rate never 100%, type of OCD is not inherited
Genes are not specific to OCD - also linked to Tourette's, autism, anorexia
Research support for genes and OFC - OCD patients and family members (genetic link) more likely to have reduced grey matter in OFC (Menzies et al)
RWA - genes may be blocked or modified, genetic explanations lull people into thinking these are simple solutions
Alternative explanations - relevance of two-process model supported by success of SD like therapy called ERP
AO1 BiA: Treating OCD
Antidepressant increase serotonin
SSRIs - prevent re-uptake of serotonin by pre-synaptic neuron
Tricyclics - block re-uptake of noradrenaline and serotonin but have more severe side-effects, so are second choice treatment
Anti anxiety drugs - BZs enhance GABA, a neurotransmitter that slows down the nervous system
D-Cycloserine - reduces anxiety
AO3 BiA: Treating OCD
Effectiveness - SSRIs better than placebo over the short term
Drug therapies are preferred - less time and less effort than CBT, and may benefit from interaction with caring doctor
Side effects - not so severe with SSRIs, more severe with tricyclics and BZs
Not a lasting cure - patients relapse when treatment stops, CBT may be preferable
Publication bias - more studies with positive results published which may bias doctor preferences
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