Immunology
Teacher recommended
?- Created by: Jenna k
- Created on: 22-02-14 11:12
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- Immunology
- Defence mechanisms
- Non-specific
- Barriers
- Protective covering (Skin)
- Epithelia covered in mucus in nose throat etc
- Hydrochloric acid in the stomach
- Phagocytosis
- Phagocytosis deals with Phagocytes
- Phagocytes are attracted to the pathogens chemo attractants along a concentration gradient
- The Phagocyte binds to the pathogen
- Lysosomes within the Phagocyte migrate towards the pathogen as the phagocyte engulfs it
- Once engulfed the Lysosomes brake down the pathogen
- The pathogen absorbs the remaining products
- Phagocytosis
- Phagocytosis deals with Phagocytes
- Phagocytes are attracted to the pathogens chemo attractants along a concentration gradient
- The Phagocyte binds to the pathogen
- Lysosomes within the Phagocyte migrate towards the pathogen as the phagocyte engulfs it
- Once engulfed the Lysosomes brake down the pathogen
- The pathogen absorbs the remaining products
- The pathogen absorbs the remaining products
- Once engulfed the Lysosomes brake down the pathogen
- Lysosomes within the Phagocyte migrate towards the pathogen as the phagocyte engulfs it
- The Phagocyte binds to the pathogen
- Phagocytes are attracted to the pathogens chemo attractants along a concentration gradient
- Phagocytosis deals with Phagocytes
- Phagocytosis
- The pathogen absorbs the remaining products
- Once engulfed the Lysosomes brake down the pathogen
- Lysosomes within the Phagocyte migrate towards the pathogen as the phagocyte engulfs it
- The Phagocyte binds to the pathogen
- Phagocytes are attracted to the pathogens chemo attractants along a concentration gradient
- Phagocytosis deals with Phagocytes
- Barriers
- Specific
- Cell mediated Response
- 1) Pathogens invade our cells
- 2) Phagocytosis occurs however instead of the pathogens product being absorbed they are displayed on the Cell-Surface membrane.
- 3) The T helper cells have receptors that compliment the antigens of the pathogens.
- 4) The T helper cell rapidly divides by Mitosis to form lots of clones. These clones become:
- Stimulants to promote phagocytosis
- Stimulate B cells to devide
- Memory B cells
- Plasma cell
- Antibodies
- 5) Kill affected cells
- Cell mediated Response
- 1) Pathogens invade our cells
- 2) Phagocytosis occurs however instead of the pathogens product being absorbed they are displayed on the Cell-Surface membrane.
- 3) The T helper cells have receptors that compliment the antigens of the pathogens.
- 4) The T helper cell rapidly divides by Mitosis to form lots of clones. These clones become:
- Stimulants to promote phagocytosis
- Stimulate B cells to devide
- Memory B cells
- Plasma cell
- Antibodies
- 5) Kill affected cells
- 4) The T helper cell rapidly divides by Mitosis to form lots of clones. These clones become:
- 3) The T helper cells have receptors that compliment the antigens of the pathogens.
- 2) Phagocytosis occurs however instead of the pathogens product being absorbed they are displayed on the Cell-Surface membrane.
- 1) Pathogens invade our cells
- Cell mediated Response
- 4) The T helper cell rapidly divides by Mitosis to form lots of clones. These clones become:
- 3) The T helper cells have receptors that compliment the antigens of the pathogens.
- 2) Phagocytosis occurs however instead of the pathogens product being absorbed they are displayed on the Cell-Surface membrane.
- 1) Pathogens invade our cells
- Humeral Response
- 1) Pathogen is engulfed by a phagocyte and the antibodies are presented on the Cell surface membrane.
- 2) T cells complement he antigens and they activate the B cells.
- 3) The B cell rapidly divides by mitosis. This produces:
- Plasma cells
- Plasma cells produce antigens rapidly
- Primary Immune Response
- Humeral Response
- 1) Pathogen is engulfed by a phagocyte and the antibodies are presented on the Cell surface membrane.
- 2) T cells complement he antigens and they activate the B cells.
- 3) The B cell rapidly divides by mitosis. This produces:
- Plasma cells
- Plasma cells produce antigens rapidly
- Primary Immune Response
- Memory B cells
- Secondary Immune Response
- Remembers the antibodies made for the next attack
- Untitled
- Plasma cells
- 3) The B cell rapidly divides by mitosis. This produces:
- 2) T cells complement he antigens and they activate the B cells.
- 1) Pathogen is engulfed by a phagocyte and the antibodies are presented on the Cell surface membrane.
- Memory B cells
- Secondary Immune Response
- Remembers the antibodies made for the next attack
- Untitled
- Plasma cells
- 3) The B cell rapidly divides by mitosis. This produces:
- 2) T cells complement he antigens and they activate the B cells.
- 1) Pathogen is engulfed by a phagocyte and the antibodies are presented on the Cell surface membrane.
- Cell mediated Response
- Non-specific
- Antibodies
- On separate document
- Vaccinations
- Passive
- Antigens are stimulated to be produce by an external source (Vaccinations)
- Active
- When our body produce antibodies on it own ocurde
- Features of Successful Vaccination Programe
- Economic and affordable vaccine
- Few side effects but work at the same time
- Must be able to be Produced, Transported and Administrated in mass quantity's
- Must be available to the majority of the population
- Why vaccinations don eliminate deseases
- timing is of the essence
- Pathogen may mutate to quickly before every one is vaccinated
- People have defective immune systems
- People may not believe in vaccinations or just not be able to get to them
- Passive
- Defence mechanisms
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