Vision

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  • Created by: SHorner96
  • Created on: 28-05-18 12:27

1. What did Spering & Carrasco (2015) study?

  • Parvocellular deficit causes dyslexia
  • The dissociation between visual perception & eye movements - eye movements can be sensitive to stimuli which we do not perceive.
  • Blindsight provides a model which integrates subcortical structures
  • Magnocellular pathway deficit impairs motor perception may cause dyslexia
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2. What did Rolfs (2015) find?

  • Patients with damage to area v1 can still display blindsight
  • Lesion to the posterior parietal cortex (dorsal pathway) causes perception deficits.
  • Eye movements actively serve perceptual and cognitive goals - saccades affect content of visual memory.
  • There is a dissociation between visual perception and eye movements

3. What does the Superior Colliculus do in vision?

  • Orients to objects
  • Controls horizontal saccades & gaze
  • Maps visual space and contains build-up, fixation and burst cells
  • Maps visual space

4. What was wrong with patient AI?

  • They could not control their saccidic impulses
  • They could not perform any eye movements but had no marked deficits - a major issue for active vision theory
  • They could not perform any eye movements but had no marked deficits - a major issue for passive vision theory
  • They were not conscious of what was in their visual fields

5. Who found that magnocellular (dorsal) pathway deficit impairs motor perception and may cause dyslexia?

  • Ajina et al., (2015)
  • Tamietto & Morrone (2016)
  • Gori et al., (2016)
  • Takemura et al., (2016)
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