thromb 0.0 / 5 ? OtherpharmUniversityNone Created by: hadarCreated on: 22-11-15 11:10 Streptokinase- mechanism of action cleaves the Arg-Val bond in plasminogen to produce active plasmin. converts inactive plasminogen molecules into active plasmin. 1 of 10 Streptokinase- therapeutic affect Plasmin in turn degrades the fibrin matrix of the thrombus- reperfusion to tissue and restoration of blood flow 2 of 10 Urokinase- mechanism of action cleaves the Arg-Val bond in plasminogen to produce active plasmin. converts inactive plasminogen molecules into active plasmin. 3 of 10 Urokinase- therapeutic affect Plasmin in turn degrades the fibrin matrix of the thrombus- reperfusion to tissue and restoration of blood flow 4 of 10 Tenecteplase- mechanism of action cleaves the Arg-Val bond in plasminogen to produce active plasmin. converts inactive plasminogen molecules into active plasmin. 5 of 10 Tenecteplase- therapeutic affect degrades the fibrin matrix of the thrombus-reperfusion to tissue and restoration of blood flow 6 of 10 Warfarin- mechanism of action inhibits vitamin K reductase- depletion of the reduced form of vitamin K 7 of 10 Warfarin- therapeutic affect decreased prothrombin levels and a decrease in the amount of thrombin generated 8 of 10 Dalteparin- mechanism potentiates the activity of ATIII, inhibiting the formation of both factor Xa and thrombin. 9 of 10 Dalteparin- therapeutic affect antithrombin binding site that is essential for high affinity binding to the plasma protein antithrombin (ATIII) 10 of 10
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