Renal Physiology 0.0 / 5 ? OtherFIUniversityNone Created by: MollyCreated on: 05-06-14 11:30 How much filtrate is reabsorbed in the proximal tubule? 60-70% 1 of 21 How much filtrate is reabsorbed in the loop of Henle? 20-255 2 of 21 K+ secretion is via which cells? Principle cells 3 of 21 K+ reabsorption is via which cells? Intercalated cells 4 of 21 What controls these processes? Aldosterone 5 of 21 Which nuclei make ADH/Vasopressin? Supraoptic and paraventricular 6 of 21 Where is ADH/Vasopressin released from? Posterior pituitary gland 7 of 21 Which receptors do ADH/Vasopressin bind to? V2 8 of 21 Where are these receptors located? The basolateral cell membranes of the principle cells in the distal part of the nephron 9 of 21 What does ADH/Vasopressin do? Increaase water and urea permeability 10 of 21 What increases ADH/Vasopressin output? Haemorrhage, increase in ECF osmolarity, sleep, fright and excerise 11 of 21 What decreases ADH/Vasopressin output? Alcohol 12 of 21 What does Aldosterone increase the reabsorption of? Na+, Cl- and water 13 of 21 What does Aldosterone increase the secretion of? K+ 14 of 21 What does Aldosterone increase the excretion of? H+ 15 of 21 What's the sensation of thirst induced by? Stimulation of hypothalamic osmoreceptors 16 of 21 Hypovolaemia is detected by what? Cardiovascular stretch receptors 17 of 21 What is pituitary (central) diabetes inspidus? No/reduced synthesis or release of ADH from hypothalamo-pituitary axis 18 of 21 What is nephrogenic (peripheral) diabetes inspidus? Lack of response by kidney to circulating ADH 19 of 21 What can an overproduction of ADH lead to? Hypervolaemia, hyponatraemia --> water intoxication 20 of 21 What are the symptoms of an overproduction of ADH? Headache, convulsions, coma and death 21 of 21
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