Neurodevelopment Hypothesis Essay Key names

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Di Forti et al 2004
Incorporates many different hypothesis/explanations of schizophrenia
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Kety et al 2002
Pps asked to track target that move sinusiodially, controls do this easily and smoothly, eye tracking in schizo more irregular
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Conrad Abebe and Austin 1991
Control pps cells are in an organised manner in those with schizo are very disorganised
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Kendall and Adams 1991
More schizophrenics born in winter/Spring
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Venables 1996
Flu associated with the cold months in Mauritius, correlated with an increased schizotypy score in the spring months. As study in Mauritius, thos increased in risk of schizo were born in september and December
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St Clair et al 1991
Famine in china between 1959 and 1961 meaning low nutrition to foetus, more schizophrenics born immediately after the famine. Supports because shows how problem during development and that need interaction with genes since not everyone developed S
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Patterson 2009
Animal models show that inducing infection in pregnant mothers (rats and mice) the offspring show behavioural abnormalities and neurochemical abnormalities that are reversed by antipsychotic drugs
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Zuckerman et al 2003
Poly IC used to mimic viral exposure because it elicits immune responses similar to those observed in viral infections
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Weinerger 1996
Weakness is the clinical data base on which it sits
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Rund 2009
Brain tissue is destroyed which results in symptoms: shown through brain scans
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Rund 2018
review shows how most evidence suggest is no neurodegeneration in schizo and states how those with the disease perform equally well in cognitive tasks before de generation
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Kety et al 2002

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Pps asked to track target that move sinusiodially, controls do this easily and smoothly, eye tracking in schizo more irregular

Card 3

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Conrad Abebe and Austin 1991

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Card 4

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Kendall and Adams 1991

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Preview of the front of card 4

Card 5

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Venables 1996

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