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6. Role of Lateral Hypothalamus in controlling eating behaviour

  • Activated by low levels of blood glucose causing feelings of hunger so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual starts eating
  • Activated by low levels of blood glucose causing feelings of satiety so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual stops eating

7. Issue of the Glucostat theory

  • It doesn't include insulin and glucagon's part in regulating blood sugar levels and therefore control of eating behaviour
  • Cannot be considered adaptive like it suggests it is as it should anticipate low levels of blood sugars instead of waiting till levels of blood sugar are low
  • It's too simplistic to suggest that this is how blood glucose is controlled

8. What is Leptin's role in controlling eating behaviour?

  • It stimulates the hypothalamus decreasing appetite
  • It's produced by fat cells and impulses are sent to the brain about the amount of fat cells present, obese rats have a high levels of leptin
  • It's produced by fat cells and impulses are sent to the brain about the amount of fat cells present, obese rats have a deficiency in leptin
  • It stimulates the hypothalamus increasing appetite

9. Which study found that when the LH was damaged it caused rats to stop eating but when it was stimulated it caused rats to overeat?

  • Heatherington and Ranson
  • Lutter et al.
  • Zhang et al.

10. Role of Ventromedial Hypothalamus

  • Activated by low levels of blood glucose causing feelings of satiety so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual stops eating
  • Activated by low levels of blood glucose causing feelings of hunger so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual starts eating

11. What did Yang et al. find?

  • when rats were injected with leptin they lost weight
  • Neuropeptide Y is produced by the hypothalamus leading to overeating then when abdominal fat is gained, the fat produces neuropeptide Y leading to more overeating
  • When Neuropeptide Y is injected into the hypothalmus of rats it causes overeating
  • When additional neuropeptide Y was injected into rat's hypothalamus it led to obesity

12. Which study found that when rats were injected with leptin they lost weight?

  • Zhang et al.
  • Yang et al.
  • Heatherington and Ranson
  • Lutter et al.