Neural Mechanisms of Controlling Eating behaviour

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1. Role of Ventromedial Hypothalamus

  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual stops eating
  • Activated by low levels of blood glucose causing feelings of hunger so the individual starts eating
  • Activated by low levels of blood glucose causing feelings of satiety so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual starts eating
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Other questions in this quiz

2. What did Wickens find?

  • When Neuropeptide Y is injected into the hypothalmus of rats it causes overeating
  • When additional neuropeptide Y was injected into rat's hypothalamus it led to obesity
  • Neuropeptide Y is produced by the hypothalamus leading to overeating then when abdominal fat is gained, the fat produces neuropeptide Y leading to more overeating

3. What did Lutter et al. find?

  • When the LH was damaged it caused rats to stop eating but when it was stimulated it caused rats to overeat
  • The hormone gherlin produced in response to stress also increases appetite
  • Neuropeptide Y is produced by the hypothalamus leading to overeating then when abdominal fat is gained, the fat produces neuropeptide Y leading to more overeating

4. Which study found that when rats were injected with leptin they lost weight?

  • Zhang et al.
  • Lutter et al.
  • Yang et al.
  • Heatherington and Ranson

5. Issue of the Glucostat theory

  • It doesn't include insulin and glucagon's part in regulating blood sugar levels and therefore control of eating behaviour
  • Cannot be considered adaptive like it suggests it is as it should anticipate low levels of blood sugars instead of waiting till levels of blood sugar are low
  • It's too simplistic to suggest that this is how blood glucose is controlled

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