Neural Mechanisms of Controlling Eating behaviour

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1. What is Leptin's role in controlling eating behaviour?

  • It's produced by fat cells and impulses are sent to the brain about the amount of fat cells present, obese rats have a deficiency in leptin
  • It's produced by fat cells and impulses are sent to the brain about the amount of fat cells present, obese rats have a high levels of leptin
  • It stimulates the hypothalamus increasing appetite
  • It stimulates the hypothalamus decreasing appetite
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2. What did Lutter et al. find?

  • The hormone gherlin produced in response to stress also increases appetite
  • Neuropeptide Y is produced by the hypothalamus leading to overeating then when abdominal fat is gained, the fat produces neuropeptide Y leading to more overeating
  • When the LH was damaged it caused rats to stop eating but when it was stimulated it caused rats to overeat

3. Which does neuropeptide Y do in terms of controlling eating behaviour?

  • It stimulates the hypothalamus increasing appetite
  • It stimulates the hypothalamus decreasing appetite
  • It's produced by fat cells and impulses are sent to the brain about the amount of fat cells present, obese rats have a high levels of leptin
  • It's produced by fat cells and impulses are sent to the brain about the amount of fat cells present, obese rats have a deficiency in leptin

4. Role of Lateral Hypothalamus in controlling eating behaviour

  • Activated by low levels of blood glucose causing feelings of hunger so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual starts eating
  • Activated by low levels of blood glucose causing feelings of satiety so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual stops eating

5. Role of Ventromedial Hypothalamus

  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual stops eating
  • Activated by low levels of blood glucose causing feelings of hunger so the individual starts eating
  • Activated by low levels of blood glucose causing feelings of satiety so the individual starts eating
  • Activated high levels of blood sugar causing feelings of satiety (fullness) so the individual starts eating

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