Neural mechanisms in eating and satiation
- Created by: Hope_Jeffries18
- Created on: 15-01-16 11:32
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- neural mechanisms in eating and satiation
- AO1:Homeos-tatis there are two systems invovled in controlling eating
- The ventromedial hypathalamus (VMH)
- initiatiates satiation when there are high glucose levels
- damage to VMH causes hyperphagia (over eating) and stimulating it inhibits eating
- A02: damaging VMH also damages paraventricul-ar node which is now belived to cause hyperphagia.
- The lateral hypthalamus (LH)
- Makes you hungry when glucose levels drop
- damge to LH in rats caused lack of eating(aphagia) stimulating LH elicits feeding behaviour
- NPY: neutransmitter Y is belived to cause obesity.
- Wickens: when injected inot rats caused to to begin feeding even when satiated
- Stanley Et al: injected rats with NPY which caused obesity in a matter of days.
- A03 Reductionist damage to LH causes deficits in other aspects of behaviour not just hunger. hunger is also controlled by neuaral circuits running through the brain
- NPY: neutransmitter Y is belived to cause obesity.
- The ventromedial hypathalamus (VMH)
- AO1 Glucose Glucose levels drop activates the LH. While an increase results the the VMH being activated
- AO1- Cognitive factors. Amygdal and the inferior prefrontal cortex
- role of the Amygdala was the selection of foods based on previous experiances.
- Rolls and Rolls found by removing the amgdala in rats caused them to consume both novel and familiar foods whearas initially they avoided novel foods.
- the role of the prefrontal cortex is to recieve messages from olfactory bulb damages=less sensory information= less eating
- role of the Amygdala was the selection of foods based on previous experiances.
- AO1:Homeos-tatis there are two systems invovled in controlling eating
- A03:- deterministic does not consider other factors ed. cultural differences
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