Genetic Control of Cell Division
- Created by: emma
- Created on: 16-04-13 15:55
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- Genetic control of cell division
- Tumour supressor genes
- Opposite to
- Proto-oncogenes
- stimulate cell division
- Normal cell?
- growth factors attach to receptor protein on CSM and 'switch on' paticular genes via relay protein in cytoplasm
- Gene mutation causes proto-oncogenes to mutate into oncogenes?
- oncogene
- receptor protein on CSM permenantley activated
- cell division is switched on, even in the absence of growth factors.
- cells divide too rapidly so tumor/cancer develops
- cell division is switched on, even in the absence of growth factors.
- code for growth factor that is produced in excessive amounts
- excessive cell division
- cells divide too rapidly so tumor/cancer develops
- excessive cell division
- receptor protein on CSM permenantley activated
- oncogene
- Normal cell?
- stimulate cell division
- Proto-oncogenes
- inhibit cell divison
- normal tumor supressor gene?
- maintain normal rates of cell division and prevent tumors
- mutated tumor supressor gene?
- inactivated!
- stops inhibiting cell division
- cell division increases
- mutant cells formed - structuarly and functionally different from normal cells
- most die
- clone selves and form tumors
- survivors...
- mutant cells formed - structuarly and functionally different from normal cells
- cell division increases
- stops inhibiting cell division
- inactivated!
- normal tumor supressor gene?
- Opposite to
- Proto-oncogenes
- stimulate cell division
- Normal cell?
- growth factors attach to receptor protein on CSM and 'switch on' paticular genes via relay protein in cytoplasm
- Gene mutation causes proto-oncogenes to mutate into oncogenes?
- oncogene
- receptor protein on CSM permenantley activated
- cell division is switched on, even in the absence of growth factors.
- cell division is switched on, even in the absence of growth factors.
- code for growth factor that is produced in excessive amounts
- excessive cell division
- excessive cell division
- receptor protein on CSM permenantley activated
- oncogene
- Normal cell?
- stimulate cell division
- Tumour supressor genes
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