Carlsson et al study review

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  • Carlsson et al
    • Aim
      • To present the current view of the relationship between schizophrenia and dopamine dysfunction
    • Procedure
      • Carlsson did not carry out his own research
      • PET is a brain imaging technique which injects the participant with a radioactive tracer that dissolves in the blood stream.
      • Some PET tracers are designed to bind to receptors on neurons in the brain. They wont be able to do this if the neurotransmitters are over-active
      • They wanted to review the methods and findings of studies looking into dopamine and schizophrenia
    • Sample
      • There is no sample
    • IV
      • There is no IV
    • DV
      • There is no DV
    • Results
      • It is unlikely that dopamine is the only dysfunctional neurotransmitter in schizophrenia
      • Dopamine change may come from some other changes in people.
      • Noradrenaline, serotonin, acetylcholine, glutamate and GABA relate to schizophrenia
    • Conclusion
      • There might be sub populations of those with schizophrenia and there might be different causes within the subpopulation
      • Glutamate deficiency leads to more positive symptoms in schizophrenia
    • Strengths and weaknesses
      • Strength - Sendt et al's review confirms carlssons suspicions of glutamate. Drugs focusing on dopamine do not work.
      • Weakness- Due to Carlsson looking into experiments with animals there is a lack of validity relating it to humans
      • Research is time locked due to it being done so long ago. It may no longer be representative
      • The research is reliable and it follows a standardised procedure


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